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PMID: 10931297 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S.

Clinical and environmental isolates of Burkholderia cepacia exhibit differential cytotoxicity towards macrophages and mast cells.

Molecular microbiology ·Vol. 36 ·No. 6 ·2000-06-00 ·Pages 1481-93

Melnikov A, Zaborina O, Dhiman N, Prabhakar BS, Chakrabarty AM, Hendrickson W

Abstract

Burkholderia cepacia is an emerging opportunistic pathogen that causes fatal infections in patients suffering from cystic fibrosis (CF) and chronic granulomatous disease. Various environmental isolates of B. cepacia are, however, capable of degrading environmental pollutants, such as trichloroethylene, 2,4,5-trichlorophenoxyacetic acid (2,4,5-T), etc., and are also highly effective in controlling plant diseases caused by nematodes and fungi. Such strains have therefore been proposed for environmental release to clean up toxic dump sites or as biopesticides. Various efforts to distinguish between clinical and environmental isolates of B. cepacia with regard to their virulence characteristics have produced ambiguous results, suggesting that newer methods are needed to test for the presence or absence of pathogenic potential in B. cepacia strains proposed for environmental release. We now report that several clinical strains of B. cepacia secrete cytotoxic factors that allow macrophage and mast cell death in the presence of external ATP. Several environmental strains had reduced activity in this regard. We also demonstrate that, while all the strains secrete enzymes that have nucleoside diphosphate kinase (Ndk), adenylate kinase (Ak) and 5'-nucleotidase activity, the level of secretion of the 5'-nucleotidase (and/or ATPase/phosphatase) appears to be lower in the environmental strains than in the clinical strains. The secretion of these enzymes is specifically activated in the presence of eukaryotic proteins such as alpha2-macroglobulin. As macrophage-or mast cell surface-associated P2Z receptors promote their cell death in the presence of mM concentrations of ATP, and as the secreted ATP-using enzymes generate various phosphorylated or non-phosphorylated adenine nucleotides that may even be better agonists than ATP in activating the P2Z receptors or may act through the activation of additional purinergic receptors, such enzymes may play an important role in allowing B. cepacia to evade host defence.

MeSH Terms
Adenosine Diphosphate/metabolism Adenosine Monophosphate/metabolism Adenosine Triphosphatases/metabolism Adenosine Triphosphate/metabolism Adenylate Kinase/metabolism Animals Burkholderia Infections/microbiology Burkholderia cepacia/isolation & purification,metabolism,pathogenicity Cell Death Cell Line Cells, Cultured Chromatography, High Pressure Liquid Cytotoxicity Tests, Immunologic Environment Female Macrophages/cytology,microbiology Male Mast Cells/cytology,microbiology Mice Mice, Inbred BALB C Nucleoside-Diphosphate Kinase/metabolism Virulence
Chemicals
Adenosine Monophosphate Adenosine Diphosphate Adenosine Triphosphate Adenylate Kinase Nucleoside-Diphosphate Kinase Adenosine Triphosphatases
Authors & Affiliations
6 authors, click to expand affiliations / ORCID
Melnikov A
Department of Microbiology and Immunology, University of Illinois College of Medicine, Chicago 60612, USA.
Zaborina O
Dhiman N
Prabhakar B S
Chakrabarty A M
Hendrickson W
Article Info
Journal
Molecular microbiology
Abbr.
Mol Microbiol
ISSN
0950-382X
Published
2000-06-00
Pages
1481-93
Language
English
Region
England
NLM ID
8712028
Subset
IM
Grants
NIDDK NIH HHS · DK 44972 · United States
NIEHS NIH HHS · ES 04050-13 · United States
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