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PMID: 12893285 Published · ppublish English

Regulation of PCNA and CAF-1 expression by the two tuberous sclerosis gene products.

Biochemical and biophysical research communications ·Vol. 307 ·No. 3 ·2003-09-04

Hengstschläger Markus, Rosner Margit, Fountoulakis Michael, Lubec Gert

Abstract

Tuberous sclerosis is an autosomal dominant tumor suppressor gene syndrome affecting about 1 in 6000 individuals. Two genes have been shown to be responsible for this disease: TSC1, encoding hamartin and TSC, encoding tuberin. A variety of tumors characteristically occur in different organs of tuberous sclerosis patients and are believed to result from defects in cell cycle/cell size control. In this study, we performed two-dimensional gel electrophoresis with subsequent mass spectrometrical identification of protein spots after overexpression of TSC1 or TSC2. We found expression of PCNA and the p48 subunit of CAF-1 to be regulated by two tuberous sclerosis gene products. CAF-1 and PCNA interact as major regulators of chromatin assembly during DNA repair. We suggest that deregulation of the control of chromatin assembly might contribute to development of tumors in tuberous sclerosis patients and provide important new insights into the molecular development, especially since deregulation of chromatin assembly and DNA repair results in genomic instability, a hallmark of tumor development.

Article Info
Journal
Biochemical and biophysical research communications
Abbr.
Biochem Biophys Res Commun
Published
2003-09-04
Indexed
2003-08-01
Updated
2012-11-15
Language
English
Country/Region
United States
NLM ID
0372516
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