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PMID: 1361523 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S. Review

Excitotoxic cell death.

Journal of neurobiology ·Vol. 23 ·No. 9 ·1992-11-00 ·Pages 1261-76

Choi DW

Abstract

Excitotoxicity refers to the ability of glutamate or related excitatory amino acids to mediate the death of central neurons under certain conditions, for example, after intense exposure. Such excitotoxic neuronal death may contribute to the pathogenesis of brain or spinal cord injury associated with several human disease states. Excitotoxicity has substantial cellular specificity and, in most cases, is mediated by glutamate receptors. On average, NMDA receptors activation may be able to trigger lethal injury more rapidly than AMPA or kainate receptor activation, perhaps reflecting a greater ability to induce calcium influx and subsequent cellular calcium overload. It is possible that excitotoxic death may share some mechanisms with other forms of neuronal death.

MeSH Terms
Amino Acids/physiology Animals Calcium/physiology Cell Death/physiology Glutamates/physiology Glutamic Acid Humans Models, Neurological Neurons/cytology
Chemicals
Amino Acids Glutamates Glutamic Acid Calcium
Authors & Affiliations
1 authors, click to expand affiliations / ORCID
Choi D W
Department of Neurology, Washington University School of Medicine, St. Louis, Missouri 63110.
Article Info
Journal
Journal of neurobiology
Abbr.
J Neurobiol
ISSN
0022-3034
Published
1992-11-00
Pages
1261-76
Language
English
Region
United States
NLM ID
0213640
Subset
IM
Grants
NINDS NIH HHS · NS 30337 · United States
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