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PMID: 15101042 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S. Research Support, U.S. Gov't, P.H.S.

Parkin gene alterations in hepatocellular carcinoma.

Genes, chromosomes & cancer ·Vol. 40 ·No. 2 ·2004-06-00 ·Pages 85-96

Wang F, Denison S, Lai JP, Philips LA, Montoya D, Kock N, Schüle B, Klein C, Shridhar V, Roberts LR, Smith DI

Abstract

The Parkin gene is an extremely large gene (1.5 Mb) within the highly unstable FRA6E common fragile site (CFS) region, which is frequently altered in ovarian, breast, and hepatocellular carcinomas. Because Parkin/FRA6E has genomic similarities to FHIT/FRA3B and WWOX/FRA16D, two other large tumor-suppressor genes that are within CFS regions, we were interested in characterizing Parkin gene alterations and their possible association with cancer. After analyzing 50 cancer-derived cell lines including 11 hepatocellular carcinoma (HCC) cell lines, we found that one HCC cell line, PLC/PRF/5, had a detectable homozygous deletion encompassing exon 3. Using quantitative duplex PCR and fluorescence in situ hybridization analysis to characterize the copy number changes of Parkin exons in HCC cell lines, we found that 4 of 11 HCC cell lines had heterozygous deletions of Parkin exons and one, Hep3B, had an exon duplication. Parkin protein expression was significantly decreased or absent in all 11 HCC cell lines. Furthermore, more than 50% of HCC primary tumors had decreased Parkin expression compared to that in normal liver tissue. Parkin gene-transfected PLC5 and Hep3B cells grew more slowly than vector-only transfectants and also showed increased sensitivity to apoptosis induced by cell-cycle inhibitors. Therefore, we suggest that Parkin may be involved in tumor suppression and that the loss of Parkin contributes to the development of hepatocarcinoma.

MeSH Terms
Apoptosis/genetics,physiology Blotting, Western/methods Carcinoma, Hepatocellular/chemistry,genetics,pathology Cell Division/genetics,physiology Chromosome Deletion DNA, Neoplasm/genetics Exons/genetics Gene Dosage Genes, Tumor Suppressor/physiology Homozygote Humans Immunohistochemistry/methods In Situ Hybridization, Fluorescence/methods Liver Neoplasms/chemistry,genetics,pathology Polymerase Chain Reaction/methods RNA, Neoplasm/genetics Transfection Tumor Cells, Cultured Ubiquitin-Protein Ligases/biosynthesis,genetics,immunology,physiology
Chemicals
DNA, Neoplasm RNA, Neoplasm Ubiquitin-Protein Ligases parkin protein
Authors & Affiliations
11 authors, click to expand affiliations / ORCID
Wang Fang
Division of Experimental Pathology, Department of Laboratory Medicine and Pathology, Mayo Clinic College of Medicine, Rochester, Minnesota, USA.
Denison Stacy
Lai Jin-Ping
Philips Leslie A
Montoya Damien
Kock Norman
Schüle Birgitt
Klein Christine
Shridhar Viji
Roberts Lewis R
Smith David I
Article Info
Journal
Genes, chromosomes & cancer
Abbr.
Genes Chromosomes Cancer
ISSN
1045-2257
Published
2004-06-00
Pages
85-96
Language
English
Region
United States
NLM ID
9007329
Subset
IM
Grants
NCI NIH HHS · CA48031 · United States
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