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PMID: 15976455 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Phospholipase D2 stimulates integrin-mediated adhesion via phosphatidylinositol 4-phosphate 5-kinase Igamma b.

Journal of cell science ·Vol. 118 ·No. Pt 13 ·2005-07-01 ·Pages 2975-86

Powner DJ, Payne RM, Pettitt TR, Giudici ML, Irvine RF, Wakelam MJ

Abstract

Cellular adhesion can be regulated by, as yet, poorly defined intracellular signalling events. Phospholipase D enzymes generate the messenger lipid phosphatidate and here we demonstrate that suppression of this reaction inhibits cellular adhesion. This effect was reversed by the addition of cell-permeable analogues of either phosphatidate or phosphatidylinositol 4,5-bisphosphate. By contrast, neither diacylglycerol nor lysophosphatidic acid were able to reverse this effect suggesting that phosphatidate itself acts directly on a target protein(s) to regulate adhesion rather than as the result of its conversion to either of these metabolite lipids. Antibodies that block beta1 and beta2 integrin-substrate interactions inhibited adhesion stimulated by both phosphatidate and phosphatidylinositol 4,5-bisphosphate indicating that these lipids regulate beta1 and beta2 integrin-mediated adhesion. In vivo, these lipids can be generated by phospholipase D2 and phosphatidylinositol 4-phosphate 5-kinase Igamma b, respectively, and over-expression of catalytically-functional forms of these enzymes dose-dependently stimulated adhesion while siRNA depletion of PLD2 levels inhibited adhesion. Furthermore the ability of over-expressed phospholipase D2 to stimulate adhesion was inhibited by a dominant-negative version of phosphatidylinositol 4-phosphate 5-kinase Igamma b. Consistent with this, phosphatidylinositol 4-phosphate 5-kinase Igamma b-mediated adhesion was dependent upon phospholipase D2's product, phosphatidate indicating that phosphatidylinositol 4-phosphate 5-kinase Igamma b is downstream of, and necessary for, phospholipase D2's regulation of adhesion. It is likely that this phospholipase D2-generated phosphatidate directly stimulates phosphatidylinositol 4-phosphate 5-kinase Igamma b to generate phosphatidylinositol 4,5-bisphosphate as this mechanism has previously been demonstrated in vitro. Thus, our data indicates that during the initial stages of adhesion, phospholipase D2-derived phosphatidate stimulates phosphatidylinositol 4-phosphate 5-kinase Igamma b to generate phosphatidylinositol 4,5-bisphosphate and that consequently this inositol phospholipid promotes adhesion through its regulation of cell-surface integrins.

MeSH Terms
Animals Cell Adhesion/drug effects,physiology Cell Line Humans Integrins/drug effects,physiology Phosphatidic Acids/pharmacology Phosphatidylinositol 4,5-Diphosphate Phosphatidylinositol Phosphates/pharmacology Phospholipase D/metabolism,pharmacology Phosphotransferases (Alcohol Group Acceptor)/chemistry,metabolism Protein Subunits/metabolism Rats Time Factors
Chemicals
Integrins Phosphatidic Acids Phosphatidylinositol 4,5-Diphosphate Phosphatidylinositol Phosphates Protein Subunits Phosphotransferases (Alcohol Group Acceptor) 1-phosphatidylinositol-4-phosphate 5-kinase PIPkin Igamma protein, rat phospholipase D2 Phospholipase D
Authors & Affiliations
6 authors, click to expand affiliations / ORCID
Powner Dale J
CRUK Institute for Cancer Studies, University of Birmingham, Edgbaston, Birmingham, B15 2TT, UK. [email protected]
Payne Roberta M
Pettitt Trevor R
Giudici M Luisa
Irvine Robin F
Wakelam Michael J O
Article Info
Journal
Journal of cell science
Abbr.
J Cell Sci
ISSN
0021-9533
Published
2005-07-01
Pages
2975-86
Language
English
Region
England
NLM ID
0052457
Subset
IM
Grants
Wellcome Trust · United Kingdom
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