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PMID: 16054045 Published · ppublish English Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S.

The hypothalamic arcuate nucleus: a key site for mediating leptin's effects on glucose homeostasis and locomotor activity.

Cell metabolism ·Vol. 1 ·No. 1 ·2005-01-00 ·Pages 63-72

Coppari R, Ichinose M, Lee CE, Pullen AE, Kenny CD, McGovern RA, Tang V, Liu SM, Ludwig T, Chua SC, Lowell BB, Elmquist JK

Abstract

Leptin is required for normal energy and glucose homeostasis. The hypothalamic arcuate nucleus (ARH) has been proposed as an important site of leptin action. To assess the physiological significance of leptin signaling in the ARH, we used mice homozygous for a FLPe-reactivatable, leptin receptor null allele (Lepr(neo/neo) mice). Similar to Lepr(db/db) mice, these mice are obese, hyperglycemic, hyperinsulinemic, infertile, and hypoactive. To selectively restore leptin signaling in the ARH, we generated an adeno-associated virus expressing FLPe-recombinase, which was delivered unilaterally into the hypothalamus using stereotaxic injections. We found that unilateral restoration of leptin signaling in the ARH of Lepr(neo/neo) mice leads to a modest decrease in body weight and food intake. In contrast, unilateral reactivation markedly improved hyperinsulinemia and normalized blood glucose levels and locomotor activity. These data demonstrate that leptin signaling in the ARH is sufficient for mediating leptin's effects on glucose homeostasis and locomotor activity.

MeSH Terms
Alleles Animals Arcuate Nucleus of Hypothalamus/metabolism Body Composition Body Weight Cell Nucleus/metabolism DNA Nucleotidyltransferases/metabolism Fertility Glucose/metabolism Green Fluorescent Proteins/metabolism Homeostasis Homozygote Hypothalamus/metabolism Immunohistochemistry Leptin/metabolism Male Mice Mice, Inbred C57BL Mice, Transgenic Models, Genetic Motor Activity Neurons/metabolism Oxygen Consumption Receptors, Cell Surface/metabolism Receptors, Leptin Signal Transduction Time Factors
Chemicals
Leptin Receptors, Cell Surface Receptors, Leptin Green Fluorescent Proteins DNA Nucleotidyltransferases FLP recombinase Glucose
Authors & Affiliations
12 authors, click to expand affiliations / ORCID
Coppari Roberto
Department of Medicine, Division of Endocrinology, Beth Israel Deaconess Medical Center, Harvard Medical School, 99 Brookline Avenue, Boston, Massachusetts 02215, USA.
Ichinose Masumi
Lee Charlotte E
Pullen Abigail E
Kenny Christopher D
McGovern Robert A
Tang Vinsee
Liu Shun M
Ludwig Thomas
Chua Streamson C
Lowell Bradford B
Elmquist Joel K
Article Info
Journal
Cell metabolism
Abbr.
Cell Metab
ISSN
1550-4131
Published
2005-01-00
Pages
63-72
Language
English
Region
United States
NLM ID
101233170
Subset
IM
Grants
NIDDK NIH HHS · DK26687 · United States
NIDDK NIH HHS · DK53301 · United States
NIDDK NIH HHS · DK57621 · United States
NIMH NIH HHS · MH61583 · United States
NIDDK NIH HHS · P01 DK56116 · United States
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