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PMID: 16595686 Published · ppublish English Journal Article Research Support, N.I.H., Extramural

Highly selective hydrolysis of fatty acyl-CoAs by calcium-independent phospholipase A2beta. Enzyme autoacylation and acyl-CoA-mediated reversal of calmodulin inhibition of phospholipase A2 activity.

The Journal of biological chemistry ·Vol. 281 ·No. 23 ·2006-06-09 ·Pages 15615-24

Jenkins CM, Yan W, Mancuso DJ, Gross RW

Abstract

Calcium-independent phospholipase A2beta (iPLA2beta) participates in numerous diverse cellular processes, such as arachidonic acid release, insulin secretion, calcium signaling, and apoptosis. Herein, we demonstrate the highly selective iPLA2beta-catalyzed hydrolysis of saturated long-chain fatty acyl-CoAs (palmitoyl-CoA approximately myristoyl-CoA >> stearoyl-CoA >> oleoyl-CoA approximately = arachidonoyl-CoA) present either as monomers in solution or guests in host membrane bilayers. Site-directed mutagenesis of the iPLA2beta catalytic serine (S465A) completely abolished acyl-CoA thioesterase activity, demonstrating that Ser-465 catalyzes both phospholipid and acyl-CoA hydrolysis. Remarkably, incubation of iPLA2beta with oleoyl-CoA, but not other long-chain acyl-CoAs, resulted in robust stoichiometric covalent acylation of the enzyme. Moreover, S465A mutagenesis or pretreatment of wild-type iPLA2beta with (E)-6-(bromomethylene)-3-(1-naphthalenyl)-2H-tetrahydropyran-2-one unexpectedly increased acylation of the enzyme, indicating the presence of a second reactive nucleophilic residue that participates in the formation of the fatty acyl-iPLA2beta adduct. Radiolabeling of intact Sf9 cells expressing iPLA2beta with [3H]oleic acid demonstrated oleoylation of the membrane-associated enzyme. Partial trypsinolysis of oleoylated iPLA2beta and matrix-assisted laser desorption ionization mass spectrometry analysis localized the acylation site to a hydrophobic 25-kDa fragment (residues approximately 400-600) spanning the active site to the calmodulin binding domain. Intriguingly, calmodulin-Ca2+ blocked acylation of iPLA2beta by oleoyl-CoA. Remarkably, the addition of low micromolar concentrations (5 microM) of oleoyl-CoA resulted in reversal of calmodulin-mediated inhibition of iPLA2 beta phospholipase A2 activity. These results collectively identify the molecular species-specific acyl-CoA thioesterase activity of iPLA2beta, demonstrate the presence of a second active site that mediates iPLA2beta autoacylation, and identify long-chain acyl-CoAs as potential candidates mediating calcium influx factor activity.

MeSH Terms
Acyl Coenzyme A/metabolism Acylation Amino Acid Sequence Animals Binding Sites Calcium/metabolism Calmodulin/metabolism Cricetinae Cricetulus Hydrogen-Ion Concentration Hydrolysis Lipid Bilayers Micelles Molecular Sequence Data Mutagenesis, Site-Directed Phospholipases A/genetics,metabolism Phospholipases A2 Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization Spodoptera
Chemicals
Acyl Coenzyme A Calmodulin Lipid Bilayers Micelles Phospholipases A Phospholipases A2 Calcium
Authors & Affiliations
4 authors, click to expand affiliations / ORCID
Jenkins Christopher M
Division of Bioorganic Chemistry and Molecular Pharmacology, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
Yan Wei
Mancuso David J
Gross Richard W
Article Info
Journal
The Journal of biological chemistry
Abbr.
J Biol Chem
ISSN
0021-9258
Published
2006-06-09
Epub
2006-00-04
Pages
15615-24
Language
English
Region
United States
NLM ID
2985121R
Subset
IM
Grants
NHLBI NIH HHS · 5P01HL57278-08 · United States
NHLBI NIH HHS · 5R01HL41250-12 · United States
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