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PMID: 17407388 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Comment

Type 2 diabetes: hypoinsulinism, hyperinsulinism, or both?

PLoS medicine ·Vol. 4 ·No. 4 ·2007-04-00 ·Pages e148

Glaser B

Abstract

暂无摘要

MeSH Terms
Adult Age of Onset Animals Birth Weight Congenital Hyperinsulinism/genetics,physiopathology Diabetes Mellitus, Type 2/blood,etiology,genetics,physiopathology Embryonic Development/genetics Female Fetal Macrosomia/etiology,genetics,physiopathology Genotype Hepatocyte Nuclear Factor 1-alpha/genetics,physiology Hepatocyte Nuclear Factor 4/deficiency,genetics,physiology Humans Hyperinsulinism/complications,congenital,embryology,physiopathology Hypoglycemia/congenital Infant, Newborn Insulin/deficiency,metabolism Insulin Resistance Insulin Secretion Islets of Langerhans/embryology,metabolism Mice Mice, Knockout Phenotype Pregnancy
Chemicals
HNF1A protein, human HNF4A protein, human Hepatocyte Nuclear Factor 1-alpha Hepatocyte Nuclear Factor 4 Hnf4a protein, mouse Insulin
Authors & Affiliations
1 authors, click to expand affiliations / ORCID
Glaser Benjamin
Endocrinology and Metabolism Service, Internal Medicine Department, Hadassah-Hebrew University Medical Center, Jerusalem, Israel. [email protected]
References (16)
16 references, click to expand
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Article Info
Journal
PLoS medicine
Abbr.
PLoS Med
ISSN
1549-1676
Published
2007-04-00
Pages
e148
Language
English
Region
United States
NLM ID
101231360
PMCID
PMC1831715
Subset
IM
Corrections
CommentOn
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