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PMID: 17785532 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Prediction of individual genetic risk to disease from genome-wide association studies.

Genome research ·Vol. 17 ·No. 10 ·2007-10-00 ·Pages 1520-8

Wray NR, Goddard ME, Visscher PM

Abstract

Empirical studies suggest that the effect sizes of individual causal risk alleles underlying complex genetic diseases are small, with most genotype relative risks in the range of 1.1-2.0. Although the increased risk of disease for a carrier is small for any single locus, knowledge of multiple-risk alleles throughout the genome could allow the identification of individuals that are at high risk. In this study, we investigate the number and effect size of risk loci that underlie complex disease constrained by the disease parameters of prevalence and heritability. Then we quantify the value of prediction of genetic risk to disease using a range of realistic combinations of the number, size, and distribution of risk effects that underlie complex diseases. We propose an approach to assess the genetic risk of a disease in healthy individuals, based on dense genome-wide SNP panels. We test this approach using simulation. When the number of loci contributing to the disease is >50, a large case-control study is needed to identify a set of risk loci for use in predicting the disease risk of healthy people not included in the case-control study. For diseases controlled by 1000 loci of mean relative risk of only 1.04, a case-control study with 10,000 cases and controls can lead to selection of approximately 75 loci that explain >50% of the genetic variance. The 5% of people with the highest predicted risk are three to seven times more likely to suffer the disease than the population average, depending on heritability and disease prevalence. Whether an individual with known genetic risk develops the disease depends on known and unknown environmental factors.

MeSH Terms
Algorithms Alleles Case-Control Studies Genetic Diseases, Inborn/genetics Genetic Predisposition to Disease Genetics, Population Genome, Human Humans Models, Genetic Polymorphism, Single Nucleotide Risk Factors
Authors & Affiliations
3 authors, click to expand affiliations / ORCID
Wray Naomi R
Genetic Epidemiology, Queensland Institute of Medical Research, Queensland 4029, Brisbane, Australia. [email protected]
Goddard Michael E
Visscher Peter M
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Article Info
Journal
Genome research
Abbr.
Genome Res
ISSN
1088-9051
Published
2007-10-00
Epub
2007-00-04
Pages
1520-8
Language
English
Region
United States
NLM ID
9518021
PMCID
PMC1987352
Subset
IM
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