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PMID: 19039135 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Genomic analysis of the clonal origins of relapsed acute lymphoblastic leukemia.

Science (New York, N.Y.) ·Vol. 322 ·No. 5906 ·2008-11-28 ·Pages 1377-80

Mullighan CG, Phillips LA, Su X, Ma J, Miller CB, Shurtleff SA, Downing JR

Abstract

Most children with acute lymphoblastic leukemia (ALL) can be cured, but the prognosis is dismal for the minority of patients who relapse after treatment. To explore the genetic basis of relapse, we performed genome-wide DNA copy number analyses on matched diagnosis and relapse samples from 61 pediatric patients with ALL. The diagnosis and relapse samples typically showed different patterns of genomic copy number abnormalities (CNAs), with the CNAs acquired at relapse preferentially affecting genes implicated in cell cycle regulation and B cell development. Most relapse samples lacked some of the CNAs present at diagnosis, which suggests that the cells responsible for relapse are ancestral to the primary leukemia cells. Backtracking studies revealed that cells corresponding to the relapse clone were often present as minor subpopulations at diagnosis. These data suggest that genomic abnormalities contributing to ALL relapse are selected for during treatment, and they point to new targets for therapeutic intervention.

MeSH Terms
B-Lymphocytes Cell Cycle/genetics Child Cyclin-Dependent Kinase Inhibitor p15/genetics Gene Deletion Gene Dosage Genes, p16 Genome, Human Genomics Humans Loss of Heterozygosity Lymphopoiesis Metabolic Networks and Pathways/genetics Mutation Oligonucleotide Array Sequence Analysis Polymorphism, Single Nucleotide Precursor B-Cell Lymphoblastic Leukemia-Lymphoma/genetics,pathology Precursor T-Cell Lymphoblastic Leukemia-Lymphoma/genetics,pathology Proto-Oncogene Proteins c-ets/genetics Recurrence Repressor Proteins/genetics
Chemicals
Cyclin-Dependent Kinase Inhibitor p15 ETS translocation variant 6 protein Proto-Oncogene Proteins c-ets Repressor Proteins
Authors & Affiliations
7 authors, click to expand affiliations / ORCID
Mullighan Charles G
Department of Pathology, St. Jude Children's Research Hospital, Memphis, TN 38105, USA.
Phillips Letha A
Su Xiaoping
Ma Jing
Miller Christopher B
Shurtleff Sheila A
Downing James R
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Article Info
Journal
Science (New York, N.Y.)
Abbr.
Science
ISSN
1095-9203
Published
2008-11-28
Pages
1377-80
Language
English
Region
United States
NLM ID
0404511
PMCID
PMC2746051
Subset
IM
Grants
NCI NIH HHS · P30 CA021765 · United States
NCI NIH HHS · P30 CA021765-30 · United States
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