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PMID: 20949590 Published · ppublish English

Cooperating G6PD mutations associated with severe neonatal hyperbilirubinemia and cholestasis.

Pediatric blood & cancer ·Vol. 56 ·No. 5 ·2011-05-26

Mizukawa Benjamin, George Alex, Pushkaran Suvarnamala, Weckbach Lana, Kalinyak KarenAnn, Heubi James E, Kalfa Theodosia A

Abstract

We report a novel glucose-6-phosphate dehydrogenase (G6PD) mutation, which we propose to name G6PD Cincinnati (c.1037A > T, p.N346I), found in combination with G6PD Gastonia (c.637G > T, p.V213L) in an infant who presented with neonatal cholestasis. The G6PD Cincinnati mutation results in a non-conservative amino acid substitution at the tetramer interface disturbing its formation, as seen by native gel electrophoresis and immunoblotting. G6PD Gastonia disrupts dimerization of the enzyme and by itself causes chronic non-spherocytic hemolytic anemia. The G6PD Cincinnati mutation may have aggravated the clinical picture of G6PD Gastonia with the result of severe perinatal hemolysis causing cholestasis and associated liver injury.

Article Info
Journal
Pediatric blood & cancer
Abbr.
Pediatr Blood Cancer
Published
2011-05-26
Indexed
2011-03-03
Updated
2016-11-22
Language
English
Country/Region
United States
NLM ID
101186624
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