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PMID: 3139430 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Selective production of interleukin 3 (IL3) and granulocyte-macrophage colony-stimulating factor (GM-CSF) in vitro by murine L3T4+ T cells: lack of spontaneous IL3 and GM-CSF production by Ly-2-/L3T4- lpr subset.

European journal of immunology ·Vol. 18 ·No. 9 ·1988-09-00 ·Pages 1367-72

Davignon JL, Kimoto M, Kindler V, De Kossodo S, Vassalli P, Izui S

Abstract

Murine spleen and lymph node L3T4+ T cells were found to spontaneously produce high levels of interleukin 3 (IL3) and granulocyte-macrophage colony-stimulating factor (GM-CSF) in cultures containing 10% fetal calf serum (FCS) in the absence of other stimulation. The IL3 and GM-CSF activities in culture supernatants peake between the fifth and seventh day of culture. The specificity of the bioassays was attested by the use of rabbit anti-IL3 and anti-GM-CSF antibodies, as well as by the detection of a maximal accumulation of IL3 and GM-CSF mRNA on the fourth day. In contrast, no significant activities of IL2, IL4 or interferon-gamma were detected in these culture supernatants. The markedly limited production of IL3 and GM-CSF in cultures performed in 1% autologous normal mouse serum and the inhibitory effect of anti-Ia or anti-L3T4 monoclonal antibody strongly suggest that the selective production of most, if not all IL3 and GM-CSF by L3T4+ T cells is a result of activation of L3T4+ T cells by fetal calf serum. All the strains of mice tested except athymic nude mice produced substantial amounts of IL3 and GM-CSF during the culture. This is in contrast to a previous report (Palacios, Eur. J. Immunol. 1984. 14: 599), indicating that only spleen cells of the MRL strain homozygous for the lpr gene spontaneously release IL3 in cultures. We found that spleen and lymph node cells from MRL/MpJ-lpr/lpr or C57BL/6J-lpr/lpr mice released, in fact, much less IL3 and GM-CSF in cultures. This was, however, due to the high proportion of the peculiar lpr Ly-2-/L3T4-T cells in spleen and lymph nodes, since after depletion of this lpr T cell subset, lymph node cells from C57BL/6J-lpr/lpr mice produced IL3 and GM-CSF at levels comparable to those in C57BL/6J-+/+ mice. These results further support the notion that the lpr Ly-2-/L3T4- T cell subset is immunologically nonfunctional and its accumulation dilutes functional L3T4+ T cells in mice bearing the lpr mutation.

MeSH Terms
Animals Antigens, Differentiation, T-Lymphocyte/analysis Antigens, Ly/analysis Blotting, Northern Cells, Cultured Colony-Stimulating Factors/biosynthesis Granulocyte-Macrophage Colony-Stimulating Factor Growth Substances/biosynthesis Histocompatibility Antigens Class II/immunology Interferon-gamma/metabolism Interleukin-2/metabolism Interleukin-3/biosynthesis,genetics Interleukin-4 Interleukins/metabolism Lymph Nodes/cytology Lymphocyte Activation Mice Mice, Inbred Strains Mice, Mutant Strains/immunology RNA, Messenger/metabolism Spleen/cytology T-Lymphocytes/classification,physiology
Chemicals
Antigens, Differentiation, T-Lymphocyte Antigens, Ly Colony-Stimulating Factors Growth Substances Histocompatibility Antigens Class II Interleukin-2 Interleukin-3 Interleukins RNA, Messenger Interleukin-4 Interferon-gamma Granulocyte-Macrophage Colony-Stimulating Factor
Authors & Affiliations
6 authors, click to expand affiliations / ORCID
Davignon J L
Department of Pathology, University of Geneva, Switzerland.
Kimoto M
Kindler V
De Kossodo S
Vassalli P
Izui S
Article Info
Journal
European journal of immunology
Abbr.
Eur J Immunol
ISSN
0014-2980
Published
1988-09-00
Pages
1367-72
Language
English
Region
Germany
NLM ID
1273201
Subset
IM
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