Home LiteratureArticle Details
PMID: 3649921 Published · ppublish English Journal Article

Heparin promotes the inactivation of antithrombin by neutrophil elastase.

Science (New York, N.Y.) ·Vol. 237 ·No. 4816 ·1987-08-14 ·Pages 777-9

Jordan RE, Kilpatrick J, Nelson RM

Abstract

Heparin is an acceleratory cofactor for antithrombin, a circulating inhibitor of blood coagulation enzymes. The presence of heparin on blood vessel walls is believed to contribute to the nonthrombogenic properties of those surfaces. In apparent opposition to this function, heparin was found to greatly accelerate the in vitro inactivation of antithrombin by neutrophil elastase. Inactivation rates in solution were potentiated several hundredfold by specific heparin fractions with anticoagulant activity. Although the data suggest that a heparin-antithrombin complex is essential for the inactivation by elastase to occur, the enzyme itself interacts tightly with heparin. These results suggest a mechanism which, if operating in vivo, could lead to a localized neutralization of the anticoagulant function of heparin at the endothelial surface.

MeSH Terms
Antithrombin III/antagonists & inhibitors,metabolism Endothelium/metabolism Heparin/metabolism,pharmacology Humans Kinetics Neutrophils/enzymology Pancreatic Elastase/metabolism
Chemicals
Antithrombin III Heparin Pancreatic Elastase
Authors & Affiliations
3 authors, click to expand affiliations / ORCID
Jordan R E
Kilpatrick J
Nelson R M
Article Info
Journal
Science (New York, N.Y.)
Abbr.
Science
ISSN
0036-8075
Published
1987-08-14
Pages
777-9
Language
English
Region
United States
NLM ID
0404511
Subset
IM
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