Effect of ventricular wall dyskinesia mode in Neurogenic Stress Cardiomyopathy (NSC) on clinical hemodynamic management.

There are various patterns of wall motion abnormalities in neurogenic stress cardiomyopathy (NSC), each exerting different impacts on hemodynamics. This study aims to investigate the effects of each wall motion abnormality pattern on hemodynamic management, thereby providing guidance for clinical management decisions. A retrospective analysis was conducted on patients diagnosed with NSC. Based on segmental ventricular wall dyskinesia, patients were categorized into four distinct groups. Clinical data were systematically collected and compared among these groups to analyze intergroup differences. In terms of EF, both the biventricular type (39.9 ± 4.3%) and the mid-ventricular type (40.7 ± 1.8%) exhibited significantly lower values compared to the apical ballooning group (46.5 ± 4.7%) (all P < 0.05). The measurements of MAPSE indicated that both the biventricular type (0.9 ± 0.2 cm) and the mid-ventricular type (0.9 ± 0.2 cm) were significantly smaller than those observed in the apical ballooning group (1.3 ± 0.3 cm) (all P < 0.05). Furthermore, the levels of NT-proBNP in the mid-ventricular type group [6843.0 (5810.0, 7705.0) pg/ml] were significantly higher than those in the apical ballooning group [3130.5 (2664.8, 4327.3) pg/ml], demonstrating a statistically significant difference (P < 0.05). Additionally, the type of ventricular wall dyskinesia was significantly correlated with the incidence of shock (P < 0.05) and the improvement time of EF (P < 0.01). Patients with NSC can exhibit various echocardiographic patterns of ventricular wall dyskinesia, each associated with distinct hemodynamic implications. This study underscores the importance of implementing individualized hemodynamic management strategies in clinical practice, tailored to the specific classification of wall motion abnormalities observed in NSC.