Autoimmunization and lymphomagenesis in parent to F1 combinations differing at the major histocompatibility complex: model for spontaneous disease caused by altered self-antigens?

Transplantation reviews ·Vol. 31 ·1976-00-00 ·Pages 156-224

Gleichmann E, Gleichmann H, Wilke W

Abstract

暂无摘要

MeSH Terms

Anemia, Hemolytic, Autoimmune/immunology Animals Antigens Autoantibodies Autoantigens B-Lymphocytes/immunology,transplantation Chronic Disease Disease Models, Animal/immunology Epitopes Glomerulonephritis/immunology Graft vs Host Reaction Histocompatibility Humans Immunization Immunosuppression Therapy Lymphadenitis/immunology Lymphoma/etiology,immunology,pathology Male Mice Mice, Inbred Strains Neoplasm Transplantation Sarcoma, Experimental/immunology T-Lymphocytes/immunology,transplantation Virus Replication

Chemicals

Antigens Autoantibodies Autoantigens Epitopes

Authors & Affiliations

3 authors, click to expand affiliations / ORCID

Gleichmann E

Gleichmann H

Wilke W

Article Info

Journal

Transplantation reviews

Abbr.

Transplant Rev

ISSN

0082-5948

DOI

10.1111/j.1600-065x.1976.tb01454.x

Published

1976-00-00

Pages

156-224

Language

English

Region

Denmark

NLM ID

0215244

Subset

External Links

PubMed source DOI: 10.1111/j.1600-065x.1976.tb01454.x

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Autoimmunization and lymphomagenesis in parent to F1 combinations differing at the major histocompatibility complex: model for spontaneous disease caused by altered self-antigens?

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