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PMID: 7499268 Published · ppublish English

Isolation of DAP3, a novel mediator of interferon-gamma-induced cell death.

The Journal of biological chemistry ·Vol. 270 ·No. 46 ·1996-01-17

Kissil J L, Deiss L P, Bayewitch M, Raveh T, Khaspekov G, Kimchi A

Abstract

Interaction of certain cytokines with their corresponding cell-surface receptors induces programmed cell death. Interferon-gamma induces in HeLa cells a type of cell death with features characteristic of programmed cell death. Here, we report the isolation of a novel gene, DAP3 (death-associated protein-3), involved in mediating interferon-gamma-induced cell death. The rescue of this gene was performed by a functional selection approach of gene cloning that is based on transfection with an antisense cDNA expression library. The antisense RNA-mediated inactivation of the DAP3 gene protected the cells from interferon-gamma-induced cell death. This property endowed the cells expressing it with a growth advantage in an environment restrictive due to the continuous presence of interferon-gamma and thus provided the basis of its selection. The gene is transcribed into a single 1.7-kilobase mRNA, which is ubiquitously expressed in different tissues and codes for a 46-kDa protein carrying a potential P-loop motif. Ectopic expression of DAP3 in HeLa cells was not compatible with cell growth, resulting in a 16-fold reduction in the number of drug-resistant stable clones. The data presented suggest that DAP3 is a positive mediator of cell death induced by interferon-gamma.

Article Info
Journal
The Journal of biological chemistry
Abbr.
J Biol Chem
Published
1996-01-17
Indexed
1996-01-17
Updated
2008-11-21
Language
English
Country/Region
United States
NLM ID
2985121R
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