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PMID: 773297 Published · ppublish English Journal Article

Killing of Torulopsis glabrata by Saccharomyces cerevisiae killer factor.

Antimicrobial agents and chemotherapy ·Vol. 9 ·No. 2 ·1976-02-00 ·Pages 352-4

Bussey H, Skipper N

Abstract

The Saccharomyces cerevisiae killer factor glycoproteins killed the pathogen Torulopsis glabrata by a mechanism involving membrane damage. Some other yeast species were unaffected by these glycoprotein toxins.

MeSH Terms
Adenosine Triphosphate/metabolism Candida/drug effects,metabolism Fungal Proteins/pharmacology Glycoproteins/pharmacology Potassium/metabolism Saccharomyces cerevisiae/drug effects Time Factors
Chemicals
Fungal Proteins Glycoproteins Adenosine Triphosphate Potassium
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Bussey H
Skipper N
References (5)
5 references, click to expand
  1. Transport of vitamin B12 in Escherichia coli: common receptor sites for vitamin B12 and the E colicins on the outer membrane of the cell envelope.
    J Bacteriol. 1973 Aug;115(2):506-13 PMID: 4579869
  2. Yeast killer factor: ATP leakage and coordinate inhibition of macromolecular synthesis in sensitive cells.
    Biochim Biophys Acta. 1973 Apr 16;298(4):868-75 PMID: 4580980
  3. Membrane-mediated killing of Saccharomyces cerevisiae by glycoproteins from Torulopsis glabrata.
    J Bacteriol. 1975 Oct;124(1):476-83 PMID: 240809
  4. Specific ion mediated chromatography of glycoproteins and neutral polysaccharides on substituted agarose gels.
    Anal Biochem. 1975 Apr;64(2):380-8 PMID: 1093441
  5. Studies on the nature of the killer factor produced by Saccharomyces cerevisiae.
    J Gen Microbiol. 1968 Apr;51(1):115-26 PMID: 5653223
Article Info
Journal
Antimicrobial agents and chemotherapy
Abbr.
Antimicrob Agents Chemother
ISSN
0066-4804
DOI
10.1128/AAC.9.2.352
Published
1976-02-00
Pages
352-4
Language
English
Region
United States
NLM ID
0315061
PMCID
PMC429527
Subset
IM
External Links
PubMed source DOI: 10.1128/AAC.9.2.352 PMC Full Text View journal details
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