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PMID: 7895506 Published · ppublish English Comparative Study Journal Article Research Support, U.S. Gov't, P.H.S.

Heterodimerization with Bax is required for Bcl-2 to repress cell death.

Current topics in microbiology and immunology ·Vol. 194 ·1995-00-00 ·Pages 331-8

Yin XM, Oltvai ZN, Korsmeyer SJ

Abstract

暂无摘要

Related Genes

Bcl-2

MeSH Terms

Amino Acid Sequence Animals Apoptosis Consensus Sequence Humans Mammals/metabolism Mice Molecular Sequence Data Mutagenesis, Site-Directed Neoplasm Proteins/chemistry,physiology Protein Conformation Protein Multimerization Protein Structure, Tertiary Proto-Oncogene Proteins/chemistry,physiology Proto-Oncogene Proteins c-bcl-2 Recombinant Fusion Proteins/metabolism Sequence Alignment Sequence Homology, Amino Acid Structure-Activity Relationship bcl-2-Associated X Protein

Chemicals

BAX protein, human Bax protein, mouse Neoplasm Proteins Proto-Oncogene Proteins Proto-Oncogene Proteins c-bcl-2 Recombinant Fusion Proteins bcl-2-Associated X Protein

Authors & Affiliations

3 authors, click to expand affiliations / ORCID

Yin X M

Howard Hughes Medical Institute, Washington University School of Medicine, St. Louis, MO 63110.

Oltvai Z N

Korsmeyer S J

Article Info

Journal

Current topics in microbiology and immunology

Abbr.

Curr Top Microbiol Immunol

ISSN

0070-217X

DOI

10.1007/978-3-642-79275-5_38

Published

1995-00-00

Pages

331-8

Language

English

Region

Germany

NLM ID

0110513

Subset

Grants

NCI NIH HHS · CA 49712-05 · United States

NHLBI NIH HHS · T32 HL07088 · United States

External Links

PubMed source DOI: 10.1007/978-3-642-79275-5_38 View journal details

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Heterodimerization with Bax is required for Bcl-2 to repress cell death.

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