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PMID: 8335898 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S.

Lymphokine-activated killer cell activity of CD4-CD8- TCR alpha beta + thymocytes.

Journal of immunology (Baltimore, Md. : 1950) ·Vol. 151 ·No. 2 ·1993-07-15 ·Pages 546-55

Arase H, Arase-Fukushi N, Good RA, Onoé K

Abstract

Functions of CD4-8-TCR alpha beta+ thymocytes, which are characterized by predominant usage of V beta 8.2 TCR, have remained unclear. In this study, we found that the CD4-8-TCR alpha beta + thymocytes expressed NK1.1 Ag and IL-2R beta-chain but not IL-2R alpha-chain. When the CD4-8- TCR alpha beta + thymocytes were cultured in the presence of IL-2, the CD4-8-TCR alpha beta + thymocytes vigorously proliferated. After 7 days of culture in the presence of 1000 U/ml of IL-2, approximately half of the CD4-8-TCR alpha beta + thymocytes lost NK1.1 Ag. However, the remaining half of the CD4-8-TCR alpha beta + cells showed increasing levels of NK1.1 Ag and acquired killer activity against tumor cells such as YAC-1, P815, and EL-4. These cells also killed syngeneic as well as allogeneic thymocytes. The LAK activity by the NK1.1+ CD4-8-TCR alpha beta + thymocytes was not inhibited by anti-NK1.1, anti-TCR alpha beta, or anti-CD44 mAb but was partially inhibited by anti-LFA-1 mAb. These findings indicate that the CD4-8-TCR alpha beta + thymocyte population can be divided into two population on the basis of NK1.1 expression after culture in the presence of IL-2. The NK1.1 Ag expression on the cultured CD4-8-TCR alpha beta + seems to be correlated to acquisition of LAK activity, although the NK1.1 Ag itself may not be directly involved in the target cell recognition. The present data suggest that the CD4-8- TCR alpha beta + thymocyte population is a functional T cell lineage which may serve as cells of immune defense and/or immune regulation.

MeSH Terms
Animals CD4 Antigens/analysis CD8 Antigens/analysis Cells, Cultured Cytotoxicity, Immunologic Interleukin-2/pharmacology Killer Cells, Lymphokine-Activated/immunology Lymphocyte Activation Mice Mice, Inbred C57BL Receptors, Antigen, T-Cell, alpha-beta/analysis Receptors, Interleukin-2/analysis Specific Pathogen-Free Organisms T-Lymphocytes/immunology Tumor Cells, Cultured
Chemicals
CD4 Antigens CD8 Antigens Interleukin-2 Receptors, Antigen, T-Cell, alpha-beta Receptors, Interleukin-2
Authors & Affiliations
4 authors, click to expand affiliations / ORCID
Arase H
Institute of Immunological Science, Hokkaido University, Sapporo, Japan.
Arase-Fukushi N
Good R A
Onoé K
Article Info
Journal
Journal of immunology (Baltimore, Md. : 1950)
Abbr.
J Immunol
ISSN
0022-1767
Published
1993-07-15
Pages
546-55
Language
English
Region
United States
NLM ID
2985117R
Subset
IM
Grants
NIA NIH HHS · AG05628-08 · United States
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