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PMID: 8614799 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Role of type I myosins in receptor-mediated endocytosis in yeast.

Science (New York, N.Y.) ·Vol. 272 ·No. 5261 ·1996-04-26 ·Pages 533-5

Geli MI, Riezman H

Abstract

Type I myosins are thought to drive actin-dependent membrane motility, but the direct demonstration in vivo of their involvement in specific cellular processes has been difficult. Deletion of the genes MYO3 and MYO5, which encode the yeast type I myosins, almost abolished growth. A double-deleted mutant complemented with a MYO5 temperature-sensitive allele (myo5-1) showed a strong defect in the internalization step of receptor-mediated endocytosis, whereas the secretory pathway remained apparently unaffected. Thus, myosin I activity is required for a budding event in endocytosis but not for several other aspects of membrane traffic.

MeSH Terms
Alleles Endocytosis/physiology Gene Deletion Mating Factor Molecular Sequence Data Mutation Myosins/genetics,physiology Peptides/metabolism Receptors, Mating Factor Receptors, Peptide/metabolism Saccharomyces cerevisiae/genetics,physiology Temperature Transcription Factors
Chemicals
Peptides Receptors, Mating Factor Receptors, Peptide Transcription Factors Mating Factor Myosins
Authors & Affiliations
2 authors, click to expand affiliations / ORCID
Geli M I
Department of Biochemistry, Biozentrum, University of Basel, Klingelbergstrasse 70, Basel, Switzerland.
Riezman H
Article Info
Journal
Science (New York, N.Y.)
Abbr.
Science
ISSN
0036-8075
Published
1996-04-26
Pages
533-5
Language
English
Region
United States
NLM ID
0404511
Subset
IM
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