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PMID: 8648295 Published · ppublish English Journal Article Research Support, U.S. Gov't, P.H.S.

K(+)- and HCO3(-)-dependent acid-base transport in squid giant axons II. Base influx.

The Journal of general physiology ·Vol. 106 ·No. 5 ·1995-11-00 ·Pages 845-62

Hogan EM, Cohen MA, Boron WF

Abstract

We used microelectrodes to determine whether the K/HCO3 cotransporter tentatively identified in the accompanying paper (Hogan, E. M., M. A. Cohen, and W. F. Boron. 1995. Journal of General Physiology. 106:821-844) can mediate an increase in the intracellular pH (pHi) of squid giant axons. An 80-min period of internal dialysis increased pHi to 7.7, 8.0, or 8.3; the dialysis fluid was free of K+, Na+, and Cl-. Our standard artificial seawater (ASW), which also lacked Na+, K+, and Cl-, had a pH of 8.0. Halting dialysis unmasked a slow pHi decrease. Subsequently introducing an ASW containing 437 mM K+ and 0.5% CO2/12 mM HCO3- had two effects: (a) it caused membrane potential (Vm) to become very positive, and (b) it caused a rapid pHi decrease, because of CO2 influx, followed by a slower plateau-phase pHi increase, presumably because of inward cotransport of K+ and HCO3- ("base influx"). Only extracellular Rb+ substituted for K+ in producing the plateau-phase pHi increase in the presence of CO2/HCO3-. Mean fluxes with Na+, Li+, and Cs+ were not significantly different from zero, even though Vm shifts were comparable for all monovalent cations tested. Thus, unless K+ or Rb+ (but not Na+, Li+, or Cs+) somehow activates a conductive pathway for H+, HCO3-, or both, it is unlikely that passive transport of H+, HCO3-, or both makes the major contribution to the pHi increase in the presence of K+ (or Rb+) and CO2/HCO3-. Because exposing axons to an ASW containing 437 mM K+, but no CO2/HCO3-, produced at most a slow pHi increase, K-H exchange could not make a major contribution to base influx. Introducing an ASW containing CO2/HCO3-, but no K+ also failed to elicit base influx. Because we observed base influx when the ASW and DF were free of Na+ and Cl-, and because the disulfonic stilbene derivatives SITS and DIDS failed to block base influx, Na(+)-dependent Cl-HCO3 exchange also cannot account for the results. Rather, we suggest that the most straightforward explanation for the pHi increase we observed in the simultaneous presence of K+ and CO2/HCO3- is the coupled uptake of K+ and HCO3-.

MeSH Terms
4,4'-Diisothiocyanostilbene-2,2'-Disulfonic Acid/pharmacology 4-Acetamido-4'-isothiocyanatostilbene-2,2'-disulfonic Acid/pharmacology Acid-Base Equilibrium/physiology Acids/metabolism Alkalies/metabolism Animals Axons/metabolism Bicarbonates/metabolism,pharmacology Biological Transport/drug effects,physiology Carbon Dioxide/pharmacology,physiology Carrier Proteins/pharmacology Cesium/pharmacology Decapodiformes Extracellular Space/metabolism Hydrogen-Ion Concentration Ion Transport/physiology Lithium/pharmacology Potassium/metabolism,pharmacology Rubidium/pharmacology Sodium/pharmacology
Chemicals
Acids Alkalies Bicarbonates Carrier Proteins Carbon Dioxide Cesium 4-Acetamido-4'-isothiocyanatostilbene-2,2'-disulfonic Acid Lithium Sodium Rubidium 4,4'-Diisothiocyanostilbene-2,2'-Disulfonic Acid Potassium
Authors & Affiliations
3 authors, click to expand affiliations / ORCID
Hogan E M
Department of Cellular and Molecular Physiology, Yale University School of Medicine, New Haven, Connecticut 06510, USA.
Cohen M A
Boron W F
Article Info
Journal
The Journal of general physiology
Abbr.
J Gen Physiol
ISSN
0022-1295
Published
1995-11-00
Pages
845-62
Language
English
Region
United States
NLM ID
2985110R
PMCID
PMC2229291
Subset
IM
Grants
NINDS NIH HHS · NS18400 · United States
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