Experimental colitis is ameliorated by inhibition of nitric oxide synthase activity.

Gut ·Vol. 38 ·No. 3 ·1996-03-00 ·Pages 475

Neilly PJ, Gardiner KR, Rowlands BJ

Abstract

暂无摘要

MeSH Terms

Animals Arginine/analogs & derivatives,therapeutic use Colitis/drug therapy Humans NG-Nitroarginine Methyl Ester Nitric Oxide/antagonists & inhibitors Nitric Oxide Synthase/antagonists & inhibitors Rats

Chemicals

Nitric Oxide Arginine Nitric Oxide Synthase NG-Nitroarginine Methyl Ester

Authors & Affiliations

3 authors, click to expand affiliations / ORCID

Neilly P J

Gardiner K R

Rowlands B J

References (4)

4 references, click to expand

Significance of systemic endotoxaemia in inflammatory bowel disease.
Gut. 1995 Jun;36(6):897-901 PMID: 7615280
Tumour necrosis factor alpha in stool as a marker of intestinal inflammation.
Lancet. 1992 Jan 11;339(8785):89-91 PMID: 1345871
Endotoxaemia and cytokine production in experimental colitis.
Br J Surg. 1995 Nov;82(11):1479-82 PMID: 8535797
Manipulation of the L-arginine-nitric oxide pathway in experimental colitis.
Br J Surg. 1995 Sep;82(9):1188-91 PMID: 7551992

Article Info

Journal

Gut

Abbr.

Gut

ISSN

0017-5749

DOI

10.1136/gut.38.3.475

Published

1996-03-00

Pages

475

Language

English

Region

England

NLM ID

2985108R

PMCID

PMC1383087

Subset

Corrections

CommentOn

PMID: 7557576

External Links

PubMed source DOI: 10.1136/gut.38.3.475 PMC Full Text View journal details

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Experimental colitis is ameliorated by inhibition of nitric oxide synthase activity.

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