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PMID: 9607768 Published · ppublish English Journal Article Research Support, Non-U.S. Gov't

Distinct roles of the co-activators p300 and CBP in retinoic-acid-induced F9-cell differentiation.

Nature ·Vol. 393 ·No. 6682 ·1998-05-21 ·Pages 284-9

Kawasaki H, Eckner R, Yao TP, Taira K, Chiu R, Livingston DM, Yokoyama KK

Abstract

The related proteins p300 and CBP (cAMP-response-element-binding protein (CREB)-binding protein)) are transcriptional co-activators that act with other factors to regulate gene expression and play roles in many cell-differentiation and signal transduction pathways. Both proteins have intrinsic histone-acetyltransferase activity and may act directly on chromatin, of which histone is a component, to facilitate transcription. They are also involved in growth control pathways, as shown by their interaction with the tumour suppressor p53 and the viral oncogenes E1A and SV40 T antigen. Here we report functional differences of p300 and CBP in vivo. We examined their roles during retinoic-acid-induced differentiation, cell-cycle exit and programmed cell death (apoptosis) of embryonal carcinoma F9 cells, using hammerhead ribozymes capable of cleaving either p300 or CBP messenger RNAs. F9 cells expressing a p300-specific ribozyme became resistant to retinoic-acid-induced differentiation, whereas cells expressing a CBP-specific ribozyme were unaffected. Similarly, retinoic-acid-induced transcriptional upregulation of the cell-cycle inhibitor p21Cip1 required normal levels of p300, but not CBP, whereas the reverse was true for p27Kip1. In contrast, both ribozymes blocked retinoic-acid-induced apoptosis, indicating that both co-activators are required for this process. Thus, despite their similarities, p300 and CBP have distinct functions during retinoic-acid-induced differentiation of F9 cells.

MeSH Terms
Apoptosis CREB-Binding Protein Cell Cycle Cell Cycle Proteins Cell Differentiation/drug effects,physiology Cell Line Cyclin-Dependent Kinase Inhibitor p21 Cyclin-Dependent Kinase Inhibitor p27 Cyclin-Dependent Kinases/antagonists & inhibitors Cyclins/biosynthesis,genetics Gene Expression Regulation Microtubule-Associated Proteins/biosynthesis,genetics Nuclear Proteins/genetics,physiology RNA, Catalytic/metabolism RNA, Messenger/metabolism Receptors, Retinoic Acid/metabolism Trans-Activators Transcription Factors/genetics,physiology Tretinoin/pharmacology Tumor Cells, Cultured Tumor Suppressor Proteins
Chemicals
Cell Cycle Proteins Cyclin-Dependent Kinase Inhibitor p21 Cyclins Microtubule-Associated Proteins Nuclear Proteins RNA, Catalytic RNA, Messenger Receptors, Retinoic Acid Trans-Activators Transcription Factors Tumor Suppressor Proteins Cyclin-Dependent Kinase Inhibitor p27 Tretinoin CREB-Binding Protein Cyclin-Dependent Kinases
Authors & Affiliations
7 authors, click to expand affiliations / ORCID
Kawasaki H
Tsukuba Life Science Center, The Institute of Physical and Chemical Research (RIKEN), Tsukuba Science City, Japan.
Eckner R
Yao T P
Taira K
Chiu R
Livingston D M
Yokoyama K K
Article Info
Journal
Nature
Abbr.
Nature
ISSN
0028-0836
Published
1998-05-21
Pages
284-9
Language
English
Region
England
NLM ID
0410462
Subset
IM
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